راهکارهای درمانی خاص در درمان لکنت زبان کلینیک تخصصی لکنت کرج بلوار سرداران-خیابان میرعماد-ابتدای گلبرگ ۲
A genetic component to stuttering
A commonly asked question of clinicians and reseachers alike is: “Does stut-tering run in families?” The short answer to this is yes, in some cases at least. Some authorities suggest that as many of 50 percent of those who stutter have a family history of the disorder (Riaz et al., 2005). Genetic research potentially offers answers as to why this perplexing disorder arises in the first place, and why it continues to develop in some individuals and not others.
There is no question that an advanced understanding of a genetic compon¬ent in stuttering is of tremendous importance, both theoretically and clinic¬ally. From a theoretical perspective, breakthroughs in this area could point toward the very causes of the disorder. We might also learn whether people who stutter with a positive history actually have identical problems to those who stutter with no history, or whether there are subtle differences. A more thorough understanding could also potentially have a huge impact on ther¬apy. Findings could help clinicians to predict whether a child is at increased risk of stuttering, and in the case of a child who is already showing signs of stuttering, the likelihood as to whether the condition will persist into adult¬hood. Ultimately, there could be the possibility of genetic engineering to reduce the number of people presenting for therapy in the first place.
Recently, Felsenfeld (2002) has pointed out that those researching a genetic explanation to speech disorders are comparative newcomers to the process of gene finding, and that stuttering specifically poses particular problems for those seeking to isolate a gene, or genes which may be responsible for the stuttering phenotype. A fundamental problem, and one we encounter throughout this book, concerns the issue of heterogeneity of the disorder. Identifying subgroups of stuttering is by no means a simple task (see the following section in this chapter for two very different interpretations) and as we have seen in chapter 1, even succinctly defining the disorder poses prob¬lems. Yet it is quite possible that subgrouping will occur as a function of increasing genetic research in this area. As